The Ischemic heart
- 其他作者: Mochizuki, Seibu.
- 出版: Boston : Kluwer Academic Publishers ©1998.
- 稽核項: 1 online resource (xliv, 575 pages) :illustrations.
- 叢書名: Progress in experimental cardiology ;1
- 標題: Myocardial Ischemia prevention & control , Coronary heart disease. , Myocardial Reperfusion Injury prevention & control , HEALTH & FITNESS , MEDICAL Cardiology. , physiopathology , Myocardial Reperfusion Injury , MEDICAL , Cardiology. , Reperfusion injury. , Myocardial Ischemia , Myocardial Ischemia physiopathology , HEALTH & FITNESS Diseases -- Heart. , Myocardial Reperfusion Injury physiopathology , Electronic books. , prevention & control , Myocardial infarction. , DiseasesHeart.
- ISBN: 0585398445 , 9780585398440
- ISBN: 079238105X , 9780792381051
- 試查全文@TNUA:
- 附註: Includes bibliographical references and index. Front Matter; Function of Cardiac Myocytes in Ischemicheart Disease: Comparative Changes in Right and Left Ventricular Cells; Remodeling of Cardiac Myocytes in Chronic Heart Disease; Hibernating Myocardium; Length-Dependent Modulation of Cardiac Muscle Contractility in Normoxia, Hypoxia, and Acidosis: Possible Implications for Ischemia; Roles of Superoxide, Nitric Oxide, and Peroxynitrite in Various Pathological Conditions; Real-Time Measurement of Nitric Oxide in Coronary Outflow during Transient Myocardial Ischemia and Reperfusion. Production of Hydrogen Peroxide During Hypoxia-Reoxygenation in Isolated MyocytesOxygen Free Radicals Enhance Ergonovine-Induced Canine Coronary Vasoconstriction; Structure and Expression of Calponin in Arterial Smooth Muscle Cells; Status of ß-Adrenoceptors, G-Proteins, and Adenylyl Cyclase in Ischemic Heart Disease; Oxygen Consumption and Mitochondrial Membrane Potential in Postischemic Myocardium; Pathophysiological Behavior of the Myocardium in Acute Ischemia and Reperfusion, with Special Emphasis on the Sarcoplasmic Reticulum. Ca2+ Ion Shifts in Vivo in Reversible and Irreversible Ischemic InjuryIonic Mechanisms of Reperfusion Injury; Mechanism of Inhibition of Na+-H+ Exchanger (NHE1) by ATP Depletion: Implications for Myocardial Ischemia; The Source and Fate of Protons in the Reperfused Ischemic Heart; Intracellular Calcium Regulation in Cardiac Cells during Acidosis; Intracellular pH Regulation and Myocardial Ischemia-Reperfusion Injury; Regulation of [Na+i and [Ca2+]i during Myocardial Ischemia and Reperfusion in a Single-Cell Model; ATP-Sensitive Potassium Channels and Myocardial Ischemia. Early and Delayed Protection Against Ventricular Arrhythmias Induced by PreconditioningEffects of Ischemic Preconditioning on Na+-Ca2+ Exchanger Activity and Ion Regulation in Isolated Perfused Rat Hearts; Long-Term but not Short-Term Cardioprotection can be Induced by Preconditioning in Hypercholesterolemia; Contribution of Endogenous Catecholamines to Preconditioning: Is it through Facilitation of Adenosine Production?; Hypoxic Preconditioning of Isolated Cardiomyocytes of Adult Rat. Pharmacological Modification of Ischemic Preconditioning During Percutaneous Transluminal Coronary AngioplastyProtection Against Injury Durin Gmyocardial Ischemia and Reperfusion: Concepts and Strategies; Mechanisms of Cardioprotective Effects of Propranolol Against Reperfusion Injury; Manganese Superoxide Dismutase Induced by Extracellular Stress Enhances Myocardial Tolerance to Ischemia-Reperfusion; Thioctic Acid Prevents Oxidativedamage in Isolated Cardiac Myocytes.
- 摘要: Over the past three decades, impressive progress in the field of pathogenesis, prevention and therapy of ischemic heart disease has resulted in a marked decline in mortality in the Western World. However, the incidence of this devastating disease is on the rise in developing countries. The Ischemic Heart is based upon a recent symposium in Tokyo on the subject. This volume is organized into two sections: (i) Pathophysiologic Mechanisms of Ischemia-Reperfusion Injury and (ii) Preconditioning and Protection of Ischemia-Reperfusion Injury, and contains up-to-date information concerning the current concepts of ischemia-reperfusion injury, the sequence of events resulting in the loss of contractile dysfunction, and mechanisms of cardioprotection by several drugs as well as the role of ischemic preconditioning in attenuating problems associated with ischemia-reperfusion injury.
- 電子資源: https://dbs.tnua.edu.tw/login?url=https://search.ebscohost.com/login.aspx?direct=true&scope=site&db=nlebk&AN=39517
- 系統號: 005296996
- 資料類型: 電子書
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- 引用網址: 複製連結
Over the past three decades, impressive progress in the field of pathogenesis, prevention and therapy of ischemic heart disease has resulted in a marked decline in mortality in the Western World. However, the incidence of this devastating disease is on the rise in developing countries. The Ischemic Heart is based upon a recent symposium in Tokyo on the subject. This volume is organized into two sections: (i) Pathophysiologic Mechanisms of Ischemia-Reperfusion Injury and (ii) Preconditioning and Protection of Ischemia-Reperfusion Injury, and contains up-to-date information concerning the current concepts of ischemia-reperfusion injury, the sequence of events resulting in the loss of contractile dysfunction, and mechanisms of cardioprotection by several drugs as well as the role of ischemic preconditioning in attenuating problems associated with ischemia-reperfusion injury.
來源: Google Book
來源: Google Book
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